PATIENT
EDUCATION MODULE
2
Coronary Artery Disease... Heart Attack
Coronary
artery disease is the process that leads to almost all heart attacks or episodes
of angina. The arteries that overly
the heart muscle and supply it with blood become plugged or blocked by the
process known as “coronary artery
disease”. The process of coronary
artery disease begins early in life. In
North American males it starts in the teens or early twenties and in women about
10 years later. The disease usually
gets slowly worse until there is enough blockage to causes symptoms or an acute
event like a heart attack.
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The
coronary arteries can be imagined like the trunk of
a maple tree (upside down) with
the thickest portion of the trunk at the top and the thinnest
branches or tributaries at the bottom.
If a blockage occurs close to the top, or trunk,
of the artery then there will be a lot of heart muscle that does not get
enough blood supply. Likewise, if a
heart attack occurs based on a blockage near the base of the trunk, then there
will likely be a lot of damage to
the heart muscle.
A
heart attack in medical terms is a ‘myocardial infarction’ (MI)
or ‘coronary thrombosis’ or ‘coronary.’ It is the result of severe
narrowing or obstruction in the coronary arteries, which blocks the blood supply
to a particular area of the heart and causes permanent
damage to that area.
The
amount of muscle damage determines the size of the heart attack.
The
most common type of heart attack is caused by coronary
thrombosis, which occurs when a clot (thrombus) blocks one or more of the
coronary arteries. Coronary thrombosis is one of the manifestations of coronary
artery disease.
Heart
attacks sometimes occur in people with little or no coronary artery disease. Spasm, or sudden constriction of the coronary artery, may also cause
a heart attack, but is relatively rare. Much
more common is the situation where there is already some degree of blockage that
is suddenly made worse by formation on a blood clot.
Symptoms of a Heart Attack
A
heart attack can come on gradually, preceded by several attacks of angina over
days, weeks, months, or even years. But a heart attack may also occur without
any apparent warning and in people who have never previously experienced any
chest pain.
Typically
you may experience any or all of the following when having a heart attack:
– discomfort or
band-like pressure, heaviness, tightness, burning or squeezing
–
indigestion, fullness or choking sensation across the chest or anywhere in the
upper body
– it may commonly
spread to the back, jaw, arms (especially the left arm)
–
it may vary in intensity from tightness to one of agonizing crushing or bursting
– it may be
continuous, or it may last a few minutes, fade, and then recur
– it may be
associated with
– sweating
– nausea and/or vomiting
– dizziness
– palpitations
– loss of consciousness
– severe weakness
– shortness of breath
Heart
attack pain is usually stronger than angina pain and does not go away when you
rest or take medication.
Reducing the Risk of Having a Heart Attack
Risk
Factors over which You Have No Control
– being male
– your age
– your family history
Risk
Factors that Can Be Altered
– cigarette smoking
– high blood pressure
– high blood cholesterol (LDL, or ‘bad’ C) & low HDL
(‘good’ C)
– obesity
– diabetes
– lack of exercise
– stress
B) Drug
Treatment. The use of drugs depends upon the nature of the heart attack and
underlying coronary disease. Antihypertensive drugs may be prescribed to lower
the blood pressure. Other drugs may be given to improve heart function, prevent
angina, or lower the level of blood cholesterol.
C) Other
Treatment. Other forms of treatment are also available. Two (invasive) ones
are
– coronary artery bypass graft
– coronary angioplasty
Their use depends on
individual needs.
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This
drawing illustrates a blockage of the left anterior descending coronary artery
on the front wall of the heart. The
area of damaged muscle, shown as a darkened area would be identified on the ECG
as an anterior or front wall heart attack.